Antibody tests may be more reliable as an indicator of HIV infection in relatively healthy groups like U.S. Army recruits. Our critics misunderstand this subject, however. Both the ELIZA and the Western Blot are antibody tests, not tests for active, replicating virus. Both have also been shown to cross-react with things other than HIV antibodies. To say as Daniel Cosgrove does that the "HIV antibody test" is confirmed by the Western Blot in many cases is merely to say that two faulty antibody tests have produced consistent results.
3) A study by Mulder et al. for the British National Research Council (published in Lancet) has been misleadingly cited by CDC officials and others as proof that a pandemic, caused by HIV, is raging through Africa. The study actually does show that, in a Ugandan village population, persons registering positive on the antibody test had a much higher death rate than antibody-negative persons, especially in the age group 25-34. What the HIV propaganda does not say is that the subjects did not die of AIDS. Of 64 deaths of persons aged 25 to 34, only 5 were diagnosed as AIDS under the very broad "Bangui" (African) definition, which requires only conditions like sustained weight loss and persistent diarrhea. Severely diseased persons are likely to have many microbes in their system, including HIV and other things that produce positive results on the inaccurate antibody tests. That this study of non-AIDS deaths was claimed to support the HIV theory of AIDS and the existence of an African AIDS pandemic is eloquent testimony to the closed mindset that rules the HIV research community.
4) A conference on nitrite inhalants (poppers), held under the auspices of the National Institute on Drug Abuse in May 1994 was attended by such HIV kingpins as Robert Gallo and Harold Jaffe-and dissenter Peter Duesberg. Participants acknowledged that the data do not support the claim that HIV is the sole or even the primary cause of Kaposi's sarcoma (KS) in gay males. This concession is particularly remarkable because KS is still officially one of the prime AIDS-defining conditions, and many gays with KS have been classified as having "HIV disease" on "presumptive" criteria that do not require antibody testing.
The problem with the HIV/KS hypothesis is that there are dozens of known cases of KS in young gay males who have never been HIV infected, and KS is very rare among non-gay HIV positives. Even the HIV stalwarts now admit that the primary cause of KS must be some agent that is specific to gay men, such as a still-undiscovered sexually transmitted microbe specific to gays, or poppers. Thus while evidence pointing to poppers as a causative factor in KS is ridiculed by the HIV propagandists, the research community is increasingly finding the evidence impossible to ignore.
5) Mounting evidence indicates that what is called "AIDS" in hemophiliacs is caused not by HIV but by the (curable) effect of foreign proteins from treatment with unpurified Factor 8, the blood coagulant that saves hemophiliacs from an early death due to prolonged internal bleeding.
The way the good news about hemophiliacs is expressed in an HIV-obsessed research culture is by lengthening the "latency period," a statistical fudge-factor that is different in every risk group and can be adjusted as necessary to explain why so many HIV-positive people are not sick. A recent British study of 111 hemophiliacs showed that so many fewer than anticipated are suffering immune system failure that 25 percent are predicted to be "AIDS free" for 20 years or more.
What is helping hemophiliacs to avoid immune deficiencies is not harmful and ineffective antiviral drugs, but new purified blood products that do not contain foreign proteins. Highly purified Factor 8 has proved so effective in protecting the immune systems of hemophiliacs that some HIV-minded researchers are thinking of employing it against "HIV disease" in non-hemophiliacs. (Complete details on the hemophiliac studies are provided in a forthcoming paper by Duesberg in Genetica.)
This use of the latency period (and mysterious "genetic immunity") to explain away the many healthy HIV positives explains, by the way, why it would prove nothing for Duesberg or anyone else to inject himself with the virus and survive. If he lived 50 years longer and died at 110, HIV science would conclude only that the latency period is sometimes unusually long, or perhaps that Duesberg was one of the lucky folk with genetic immunity. (As for ourselves, we stick to scientific evidence and have no interest in showmanship.)
We find a similar use of this convenient fudge factor in Daniel Cosgrove's argument that the latency period accounts for why nearly 90 percent of AIDS cases are still male in North America and Europe, despite changes in the definition of the syndrome aimed at including more women. That excuse gets thinner every year as confident predictions based upon the "everyone is at risk" ideology fail to come true. We quote again the important finding of the National Research Council: "The convergence of evidence shows that the HIV/AIDS epidemic is settling into spatially and socially isolated groups and possibly becoming endemic within them." This is the opposite of what the HIV theory predicted.
6) The Tenth Annual International AIDS Conference in Yokohama in August 1994 was the last of its kind. The annual gathering of the multitudes who make their living from HIV will be skipped next year, because HIV science is at a virtual standstill and there is nothing of importance to announce. The great breakthrough touted in Yokohama was a study claiming a reduction in the rate of infant HIV infection when expectant mothers and babies were given AZT. The study was terminated abruptly, as has happened with earlier AZT studies that began to show favorable results at an early stage.
In consequence a highly toxic drug that is known to be ineffective and positively harmful in antibody-positive adults will be given routinely to unborn and newborn infants with antibody-positive mothers, although most of these infants would never be HIV infected anyway. The uncertain benefit and great risk would make such a reckless course of conduct unimaginable in normal circumstances, but the AZT lobby backed by HIV hysteria has the power to overrule the prudent standards that protect the public from other dangerous and unproven drugs.
What the Yokohama conference lacked in science it more than made up in scare stories and plans for worldwide social engineering to protect Asia and Africa from depopulation. (As Tom Bethell says, AIDS is not so much a disease as an open-ended government program.) The media as usual reported uncritically the claims that HIV infection is increasing rapidly everywhere in Asia and Africa and that the relatively low number of AIDS cases actually reported is just the tip of the iceberg.
What the media did not report is that the same virus that is supposed to be newly infecting millions of people every year in regions where reliable statistics are hard to come by has been stable in the U.S. population ever since testing began, almost 10 years ago. Every year the AIDS agencies ominously report that "1 million people are now infected with HIV," and the reporters never point out that the figure was exactly the same the year before and the year before that. We call upon the CDC to confirm or deny what The New York Times reported months ago-which is that the most reliable studies show that the actual total number of antibody-positive Americans is well under one million, and not rising. The thoroughly researched American figures utterly contradict the inflated claims about HIV infection rates in Asia and Africa-unless we indulge the kind of racist speculation about non-white sexual habits that Charles Geshekter rightly denounces in his letter.
As one of us (Mullis) has frequently commented, there is no paper in the scientific literature that reviews all the relevant evidence and establishes that HIV is the cause of AIDS. George Fergus to the contrary notwithstanding, the original papers announcing the discovery of HIV in 1984 said only that HIV had been found in some (not all) of a small group of AIDS patients. No proof was presented, but Dr. Gallo and his fellow virologists seemed so confident that the research community wrongly assumed that they had the proof. Once that assumption was set in concrete as the foundation for funding, critics could no longer be tolerated.
Instead of a paper that takes the objections seriously and undertakes to prove the point at issue, we find only polemics that assume the HIV theory and defend it against specific criticisms with question-begging arguments. Mark Nowak, for example, says that the failure of HIV to cause AIDS in the many chimps who have been deliberately infected with the virus says nothing about whether HIV causes AIDS in humans. Our point was that one of the recognized ways of proving that a virus does cause a disease in humans is to cause the disease in animals by infecting them with the virus, and such efforts have failed with HIV. The chimps do get HIV infection, just like humans, and HIV does no damage to their immune systems. Why not? To say as Mark Nowak and the HIV scientists do that "HIV does not cause AIDS in chimps" is merely to restate the question.
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