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What Causes AIDS?

It's an open question.

(Page 2 of 3)

The claimed correlation between HIV and AIDS is flawed at an even more fundamental level, however. Even if the "AIDS test" were administered in every case, the tests are unreliable. Authoritative papers in both Bio/Technology (June 1993) and the Journal of the American Medical Association (November 27, 1991) have shown that the tests are not standardized and give many "false positives" because they react to substances other than HIV antibodies. Even if that were not the case, the tests at best confirm the presence of antibodies and not the virus itself, much less the virus in an active, replicating state. Antibodies typically mean that the body has fought off a viral infection, and they may persist long after the virus itself has disappeared from the body. Since it is often difficult to find live virus even in the bodies of patients who are dying of AIDS, Gallo and others have to speculate that HIV can cause AIDS even when it is no longer present and only antibodies are left.

Just as there are cases of AIDS without HIV, there are cases of HIV-positive persons who remain healthy for over a decade and who may never suffer from AIDS. According to Warner C. Greene's article in Scientific American, "It is even possible that some rare strains [of HIV] are benign. Some homosexual men in the U.S. who have been infected with HIV for at least 11 years show as yet no signs of damage to their immune systems. My colleagues�and I are studying these long-term survivors to ascertain whether something unusual about their immune systems explains their response or whether they carry an avirulent strain of the virus."

The faulty correlation between HIV and AIDS would not disprove the HIV theory if there were strong independent evidence that HIV causes AIDS. As we have seen, however, researchers have been unable to establish a mechanism of causation. Nor have they succeeded in confirming the HIV model by inducing AIDS in animals. Chimps have repeatedly been infected with HIV, but none of them have developed AIDS. In the absence of a mechanism or an animal model, the HIV theory is based only upon a correlation that turns out to be primarily an artifact of the theory itself.

In light of the importance of the correlation argument, it is astonishing that no controlled studies have been done for three of the major risk groups: transfusion recipients, hemophiliacs, and drug abusers. Two ostensibly controlled studies involving men's groups in Vancouver and San Francisco purportedly show that AIDS developed only in the HIV-positive men and never in the "control group" of HIV negatives. These studies were designed not to test the HIV theory but to measure the rate at which HIV-positive gay men develop AIDS. They did not compare otherwise similar persons who differ only in HIV status, did not control effectively for drug use, and did not fully report the incidence of AIDS-defining diseases in the HIV-negative men. The research establishment accepted these studies uncritically because they give the HIV theory some badly needed support. But the main point they supposedly prove has already been thoroughly disproved: AIDS does occur in HIV-negative persons.

According to the official theory, HIV is a virus newly introduced into the American population, which has had no opportunity to develop any immunity. It follows that viral infection should spread rapidly, moving from the original risk groups (gays, drug addicts, transfusion recipients) into the general population. This is what the government agencies confidently predicted, and AIDS advertising to this day emphasizes the theme that "everyone is at risk."

The facts are otherwise. AIDS is still confined mainly to the original risk groups, and AIDS patients in the United States are still almost 90-percent male. Health-care workers, who are constantly exposed to blood and bodily fluids of AIDS patients, have no greater risk of contracting AIDS that the population at large. Among millions of health-care workers, the CDC claims only seven or eight (poorly documented) cases of AIDS supposedly developed through occupational exposure. By contrast, the CDC estimates that accidental needle sticks lead to more than 1,500 cases of hepatitis infection each year. Even prostitutes are not at risk for AIDS unless they also use drugs.

Far from threatening the general heterosexual population, AIDS is confined mainly to drug users and gay men in specific urban neighborhoods. According to a 1992 report by the prestigious U.S. National Research Council, "The convergence of evidence shows that the HIV/AIDS epidemic is settling into spatially and socially isolated groups and possibly becoming endemic within them." This factual picture is so different from what the theory predicts, and so threatening to funding, that the AIDS agencies have virtually ignored the National Research Council report and have continued to preach the fiction that "AIDS does not discriminate."

Not only is AIDS mostly confined to isolated groups in a few U.S. cities, but HIV infection is not increasing. Although a virus newly introduced to a susceptible population should spread rapidly, for several years the CDC has estimated that a steady 1 million Americans are HIV positive. Now it appears that the figure of 1 million is finally about to be revised--downward. According to a story by Lawrence Altman in the March 1 New York Times, new statistical studies indicate that only about 700,000 Americans are HIV positive, and the official estimate will accordingly be reduced sometime this summer.

While HIV infection remains steady at this modest level in the United States, World Health Organization officials claim that the same virus is spreading rapidly in Africa and Asia, creating a vast "pandemic" that threat ens to infect at least 40 million people by the year 2000, unless billions of dollars are provided for prevention to the organizations sounding the alarm. These worldwide figures, especially from Africa, are used to maintain the thesis that "everyone is at risk" in the United States. Instead of telling Americans that AIDS cases here are almost 90-percent male, for example, authorities say that worldwide the majority of AIDS sufferers are female. With the predictions of a mass epidemic in America and Europe failing so dramatically, AIDS organizations rely on the African figures to vindicate their theory.

But these African figures are extremely soft, based almost entirely on "clinical diagnoses," without even inaccurate HIV testing. What this means in practice is that Africans who die of diseases that have long been common there--especially wasting disease accompanied by diarrhea--are now classified as AIDS victims. Statistics on "African AIDS" are thus extremely manipulable, and witnesses are emerging who say that the epidemic is greatly exaggerated, if it exists at all.

In October 1993, the Sunday Times of London reported on interviews with Philippe and Evelyne Krynen, heads of a 230-employee medical relief organization in the Kagera province of Tanzania. The Krynens had first reported on African AIDS in 1989 and at that time were convinced that Kagera in particular was in the grip of a vast epidemic. Subsequent years of medical work in Kagera have changed their minds. They have learned that what they had thought were "AIDS orphans" were merely children left with relatives by parents who had moved away and that HIV-positive and HIV-negative villagers suffer from the same diseases and respond equally well to treatment. Philippe Krynen's verdict: "There is no AIDS. It is something that has been invented. There are no epidemiological grounds for it; it doesn't exist for us."

Krynen's remark calls attention to the fact that AIDS is not a disease. Rather, it is a syndrome defined by the presence of any of 30 separate and previously known diseases, accompanied by the actual or suspected presence of HIV. The definition has changed over time and is different for Africa (where HIV testing is rare) than for Europe and North America. The official CDC definition of AIDS in the United States was enormously broadened for 1993 in order to distribute more federal AIDS money to sick people, especially women with cervical cancer. As a direct result, AIDS cases more than doubled in 1993. Absent the HIV mystique, there would be no reason to believe that a single factor is causing cervical cancer in women, Kaposi's sarcoma in gay males, and slim disease in Africans.

The HIV paradigm is failing every scientific test. Research based upon it has failed to provide not only a cure or vaccine but even a theoretical explanation for the disease-causing mechanism. Such success as medical science has had with AIDS has come not from the futile attempts to attack HIV with toxic antiviral drugs like AZT but from treating the various AIDS-associated diseases separately. Predictions based on the HIV theory have been falsified or are supported only by dubious statistics based mainly on the theory itself. Yet the HIV establishment continues to insist that nothing is wrong and to use its power to exclude dissenting voices, however eminent in science, from the debate.

Like other leaders of the scientific establishment, Nature Editor John Maddox is fiercely protective of the HIV theory. He indignantly rejected a scientific paper making the same points as this article. When Duesberg first argued his case in 1989 in the prestigious Proceedings of the National Academy of Science, the editor promised that his paper would be answered by an article defending the orthodox viewpoint. The response never came. The editors of the leading scientific journals have refused to print even the brief statement of the Group for the Scientific Reappraisal of the HIV/AIDS Hypothesis, which has over 300 members. The statement notes simply that "many biomedical scientists now question this hypothesis" and calls for "a thorough reappraisal of the existing evidence for and against this hypothesis."

Such a reappraisal would include the following elements: Genuinely controlled epidemiological studies of all the major risk groups: homosexuals, drug users, transfusion recipients, and hemophiliacs. The studies should employ an unbiased definition of AIDS. Too often we have been told that HIV always accompanies AIDS, only to learn that this is so because AIDS without HIV is named something else. The studies should be performed by persons who are committed to investigating the HIV theory rather than defending it. There is reason to suspect that properly controlled studies of transfusion recipients and hemophiliacs in particular will show that the incidence of AIDS-defining diseases is independent of HIV status.

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